 |
| Syringeosphere |
Obviously they compare one SARS-CoV-2 virus to another SARS-CoV-2 virus.
But which ones?
One way is to compare the Wuhan, China version of the virus discovered on December 30, 2019, to subsequent SARS-CoV-2 viruses discovered later around the world:
"In addition to understanding the transmissibility and pathogenicity of these emerging variants, it is crucially important to characterize their antigenicity and the level of cross-protection provided by infection by earlier viruses that are genetically and antigenically similar to the virus that first emerged in December 2019 and which is used in all of the current vaccine preparations."
(SARS-CoV-2 variants, emphasis added). That method is used in many published papers.
But, in today's post I am comparing, country by country, SARS-CoV-2 viruses to the original Wuhan, China SARS-CoV-2 virus (appendices: a, b, c, de, fg, hj, i, klqnovwxyz, m, p, rs, t, u) [the scrambling of letters is to throw off hoaxycloroxadope users].
Those appendices containing those results can be compared to the previous appendices located in other posts (such as the "On The Origin of Genieology - 4" link above).
As I have indicated previously, the chaotic changes revealed in these appendices are indicative of the damage done by the Big Pharma war on anything that moves, more so than on the 'magic words' of pop Genieology (see On The Origin Of The Home Of COVID-19, 2, 3, 4, 5, 6, 7, 8, 9, 10, 11, 12, 13, 14, 15, 16, 17, 18, 19, 20, 21, 22, 23, 24, 25, 26, 27).
In that series and others I have pointed out that the scene is like an accident or tornado damaged site with parts mixed with other good and bad parts.
New research supports that hypothesis indicating that mutualist microbes in us can be partially damaged but continue to replicate viruses, or worse:
"An unresolved issue of SARS-CoV-2 disease is that patients often remain positive for viral RNA as detected by PCR many weeks after the initial infection in the absence of evidence for viral replication. We show here that SARS-CoV-2 RNA can be reverse-transcribed and integrated into the genome of the infected cell and be expressed as chimeric ['formed from parts of various animals'] transcripts fusing viral with cellular sequences. Importantly, such chimeric ['formed from parts of various animals'] transcripts are detected in patient-derived tissues. Our data suggest that, in some patient tissues, the majority of all viral transcripts are derived from integrated ['with various parts or aspects linked'] sequences. Our data provide an in-sight into the consequence of SARS-CoV-2 infections that may help to explain why patients can continue to produce viral RNA after recovery." [killing microbe hosts of SARS-CoV-2 with antibiotics will do that too]
(Reverse-transcribed
SARS-CoV-2 RNA can integrate into the genome of cultured human cells
and can be expressed in patient-derived tissues). It sounds like an accident scene, which is why I recently suggested that virologists take accident reconstruction classes (Inside Job and/or Conspiracy?).
Things are getting curioser [magic-er] and curiouser [magic-er] ("the cause for the prolonged and recurrent production of viral RNA remains unknown" - ibid at Reverse-transcribed SARS-CoV-2 RNA can integrate into the genome of cultured human cells and can be expressed in patient-derived tissues; cf. No evidence of SARS-CoV-2 reverse transcription and integration as the origin of chimeric transcripts in patient tissues and Response to Parry et al.: Strong evidence for genomic integration of SARS-CoV-2 sequences and expression in patient tissues).
The next post in this series is here, previous post in this series is here.
A friendly virus reports back to us about what it is like to enter into a vastly larger single-celled host "to take control over its highly complex machines" (The New Paradigm: The Physical Universe Is Mostly Machine).
No comments:
Post a Comment